Consider this: The elite would never release a plague without an easy cure, and along with this ebola outbreak an American biowarfare firm has been working in Sierra Leon for the last five years. Google that. Sierra Leon has actually identified them as the perpetrators of this outbreak and kicked them out of the country. There is absolutely no doubt this outbreak was intentionally caused by the U.S. war department.
‘The scientists, led by Thomas Geisbert at Boston University, used a relatively new genomics technique called RNA interference to defeat the virus. Here’s how it works. First, a little background: the Ebola virus is made of RNA, just like the influenza virus. And just like influenza, Ebola has very few genes – only 8. One of its genes, called L protein, is responsible for copying the virus itself. Two others, called VP24 and VP35, interfere with the human immune response, making it difficult for our immune system to defeat the virus.Geisbert and his colleagues (including scientists from Tekmira Pharmaceuticals and USAMRIID) designed and synthesized RNA sequences that would stick to these 3 genes like glue. How did they do that? We know the Ebola genome’s sequence – it was sequenced way back in 1993. And we know that RNA sticks to itself using the same rules that DNA uses. This knowledge allowed Geisbert and colleagues to design a total of 10 pieces of RNA (called “small interfering RNA” or siRNA) that they knew would stick to the 3 Ebola genes. They also took care to make sure that their sticky RNA would not stick to any human genes, which might be harmful. They packaged these RNAs for delivery by inserting them into nanoparticles that were only 81-85 nanometers across.
In the key experiment, the scientists infected rhesus monkeys with a dose of Ebola that was 30,000 times greater than the normal fatal dose. They injected the siRNA treatments 30 minutes later, and again each day for 6 days. All the monkeys survived with no long-term effects.
And if it is intentional, a cure is known. There would simply be no other way to do business. Here is the treatment, complete with MOA. This is a treatment and not a cure, your immune system wipes out the virus, and the treatment gives your immune system time to do it. Here is what Ebola does that is fatal: It causes the complete removal of all vitamin C from the body. No one actually knows what mechanism is involved in doing this, other than a malfunction that is not permanently destructive to whatever is triggered to remove all vitamin C. All the researchers know is that vitamin C drops to zero and all the symptoms of ebola are consistent with a complete loss of vitamin C.
How do I know this? A doctor who has remained anonymous and has worked with ebola victims has discovered this, at last check this cannot be googled which confirms this doctor did not just copy paste, SO POST IT EVERYWHERE; GET THIS OUT THERE, THE TREATMENT FOR EBOLA WHICH WILL PREVENT DEATH IS KNOWN AND THIS IS AN EMERGENCY REQUEST FOR MY READERS TO SPREAD THIS INFO AND STOP THIS EBOLA ATTACK IN ITS TRACKS.
From an anonymous doctor:Summary:
"The very first symptoms of ebola are exactly the same as scurvy, which is caused by inadequate vitamin C. Though scurvy is seldom fatal as a primary condition, scurvy also represents only a partial deficiency of vitamin C, the body still has a LOT of vitamin C compared to zero, which ebola causes. Absent ANY vitamin C, blood vessels become very weak and start to lose blood, and platelets become ineffective and unable to trigger clots. So death by ebola is caused by massive internal bleeding and loss of blood, which can be stopped simply by taking enormous doses of vitamin C until the immune system succeeds in killing off the virus."Begin:
Ebola is probably the best known of a class of viruses known as hemorrhagic fever viruses. In fact, Ebola virus was initially recognized in 1976. Other less known but related viral syndromes include yellow fever, dengue hemorrhagic fever, Rift Valley fever, Crimean-Congo hemorrhagic fever, Kyasanur Forest disease, Omsk hemorrhagic fever, hemorrhagic fever with renal syndrome, Hantavirus pulmonary syndrome, Venezuelan hemorrhagic fever, Brazilian hemorrhagic fever, Argentine hemorrhagic fever, Bolivian hemorrhagic fever, and Lassa fever. The Ebola virus infection, also known as African hemorrhagic fever, has the distinction of having the highest case-fatality rate of the viral infections noted above, ranging from 53% to 88%. These viral hemorrhagic fever syndromes share certain clinical features. The Cecil Textbook of Medicine notes that these diseases are characterized by capillary fragility, which translates to easy bleeding, that can frequently lead to severe shock and death. These diseases also tend to consume and/or destroy the platelets, which play an integral role in blood clotting. The clinical presentation of these viral diseases is similar to scurvy, which is also characterized by capillary fragility and a tendency to bleed easily. Characteristic skin lesions develop, which are actually multiple tiny areas of bleeding into the skin that surround the hair follicles. some cases even include bleeding into already healed scars.In the classic form of scurvy that evolves very slowly from the gradual depletion of vitamin C body stores, the immune system will be sufficiently compromised for infection to claim the patient’s life before the extensive hemorrhage that occurs after all vitamin C stores have been completely exhausted. Ebola virus and the other viral hemorrhagic fevers are much more likely to cause hemorrhaging before any other fatal infection has a chance to become established. This is because the virus so rapidly and totally metabolizes and consumes all available vitamin C in the bodies of the victims that an advanced stage of scurvy is literally produced after only a few days of the disease.
The scurvy is so complete that the blood vessels generally cannot keep from hemorrhaging long enough to allow an infective complication to develop. Also, the viral hemorrhagic fevers typically only take hold and reach epidemic proportions in those populations that would already be expected to have low body stores of vitamin C, such as is found in many of the severely malnourished Africans. In such individuals, an infecting hemorrhagic virus will often wipe out any remaining vitamin C stores before the immune systems can get the upper hand and initiate recovery. When the vitamin C stores are rapidly depleted by large infecting doses of an aggressive virus, the immune system gets similarly depleted and compromised. However, this point is largely academic after hemorrhaging throughout the body has begun.
To date, no viral infection has been demonstrated to be resistant to the proper dosing of vitamin C as classically demonstrated by Klenner. However, not all viruses have been treated with Klenner-sized vitamin C doses, or at least the results have not been published. Ebola viral infection and the other acute viral hemorrhagic fevers appear to be diseases that fall into this category. Because of the seemingly exceptional ability of these viruses to rapidly deplete vitamin C stores, even larger doses of vitamin C would likely be required in order to effectively reverse and eventually cure infections caused by these viruses.
Cathcart (1981), who introduced the concept of bowel tolerance to vitamin C discussed earlier, hypothesized that Ebola and the other acute viral hemorrhagic fevers may well require 500,000 mg of vitamin C daily to reach bowel tolerance! Whether this estimate is accurate, it seems clear as evidenced by the scurvy-like clinical manifestations of these infections that vitamin C dosing must be vigorous and given in extremely high doses. If the disease seems to be winning, then even more vitamin C should be given until symptoms begin to lessen. Obviously, these are viral diseases that would absolutely require high doses of vitamin C intravenously as the initial therapy. The oral administration should begin simultaneously, but the intravenous route should not be abandoned until the clinical response is complete. Death occurs too quickly with the hemorrhagic fevers to be conservative when dosing the vitamin C. (from Vitamin C, Infectious Diseases, and Toxins:Curing the Incurable by Thomas E. Levy MD JD)
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